ENDOTHELIAL DYSFUNCTION AND IMMUNE–FIBROTIC CROSSTALK AS CENTRAL DRIVERS OF POST-COVID PROGRESSION IN RHEUMATIC DISEASES
DOI:
https://doi.org/10.5281/zenodo.18181001Abstract
Endothelial dysfunction has emerged as one of the most consistent and clinically significant consequences of COVID-19, persisting well beyond viral clearance. The vascular endothelium plays a pivotal role in immune regulation, tissue perfusion, and connective tissue homeostasis. In rheumatic diseases—including ankylosing spondylitis, rheumatoid arthritis, reactive arthritis, osteoarthritis, allergic vasculitis, and systemic sclerosis—endothelial impairment represents a shared pathogenic pathway that contributes to chronic inflammation, fibrosis, and joint destruction.
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